[1] Kozopas KM, Yang T, Buchan HL, et al. MCL1, a gene expressed in programmed myeloid cell differentiation, has sequence similarity to BCL-2 [J]. PNAS, 1993, 90(3):3516-3520.
[2] Rahamani M, Davis EM, Bauer C, et al. Apoptosis induced by the kinase inhibitor BAY 43-9006 in human leukemia cells involves down-regulation of Mcl-1 through inhibitor of translation [J]. J Biol Chem, 2005, 280(42):35217-35227.
[3] 冯楠, 张学军. Mcl-1 基因在肿瘤靶向治疗中的研究进展 [J].
中华临床医师杂志, 2009, 3(11):1883-1887.
[4] 王加连. 蟾蜍的药用价值及人工养殖研究 [J]. 江苏农业科学 杂志, 2009, 1 :240-241.
[5] 吉晓霞, 谭晖. Mcl-1 基因及其在白血病中的作用 [J]. 国际病 理科学与临床杂志, 2009, 29(1):64-68.
[6] Nijhawan D, Tang M, Traer E, et al. Elimination of Mcl-1 is required for the initiation of apoptosis following ultraviolet irradiation [J].
Genes Dev, 2003, 17(12):1475-1486.
[7] Maurer U, Charvet C. Glycogen synthase kinase-3 regulates mitochondrial outer membrane permeabilization and apoptosis by destabilization of MCL-1 [J]. Mol Cell, 2006, 21(6):728-730.
[8] Kobayashi S, Lee SH. Serine 64 phosphorylation enhances the antiapoptotic function of Mcl-1 [J]. J Biol Chem, 2007, 282(25): 18407-18417.
[9] Chetoui N, Sylla K. Down-regulation of mcl-1 by small interfering RNA sensitizes resistant melanoma cells to fas-mediated apoptosis [J]. Mol Cancer Res, 2008, 6(1):42-52.
[10] Song L, Coppola D, Livingston S, et al. Mcl-1 regulates survival and sensitivity to diverse apoptotic stimuli in human non-small cell lung cancer cells [J]. Cancer Biol Ther, 2005, 4(3):267-276.
[11] Wuillme TS, Robillard N, Gomez P, et al. Mcl-1 is over expressed in multiple myeloma and associated with relapse and shorter survival [J]. Leukemia, 2005, 19(7):1248-1252.
[12] Schulze BH, Fleischer B, Schuchmann M, et al. Suppression of Mcl-1 via RNA interference sensitizes human hepatocellular carcinoma cells towards apoptosis induction [J]. BMC Cancer, 2006, 6(232):1-14.
[13] 张阳德, 廖明媚, 赵劲风, 等. Mcl-1 基因与肝细胞癌 [J]. 中 国现代医学杂志, 2008, 18(7):906-910.
[14] Yoon JH, Werneburg NW, Higuchi H, et al. Bile acids inhibit Mcl-1 protein turnover via an epidermal growth factor receptor/Raf-1 -dependent mechanism [J]. Cancer Res, 2002, 62(22):6500- 6505. |