[1] Goodman DS. Vitamin A and retinoids in health and disease[J]. N Engl J Med, 1984, 310:1023-1031. [2] Balmer JE, Blomhoff R. Gene expression regulation by retinoic acid[J]. J Lipid Res, 2002, 43(11):1773-1808. [3] Felipe F, Mercader J, Ribot J, et al. Effects of retinoic acid admini-stration and dietary vitamin A supplementation on leptin expression in mice:lack of correlation with changes of adipose tissue mass and food intake[J]. Biochim Biophys Acta, 2005, 1740(2):258-265. [4] Berry DC, Noy N. All-trans-retinoic acid represses obesity and insu-lin resistance by activating both peroxisome proliferation-activated receptor β/δ and retinoic acid receptor[J]. Mol Cell Biol, 2009, 29(12):3286-3296. [5] Glover J. Factors affecting vitamin-a transport in animals and man[J]. Proc Nutr Soc, 1983, 42(1):19-30. [6] Ransom J, Morgan PJ, McCaffery PJ, et al. The rhythm of retinoids in the brain[J]. J Neurochem, 2014, 129(3):366-376. [7] Zhang EE, Liu Y, Dentin R, et al. Cryptochrome mediates circadian regulation of cAMP signaling and hepatic gluconeogenesis[J]. Nat Med, 2010, 16(10):1152-1156 [8] Kastner P, Mark M, Ghyselinck N, et al. Genetic evidence that the retinoid signal is transduced by heterodimeric RXR/RAR functional units during mouse development[J]. Development, 1997, 124(2):313-326. [9] Mangelsdorf DJ, Evans RM. The RXR heterodimers and orphan receptors[J]. Cell, 1995, 83:841-850. [10] Wolf G, Lane MD, Johnson BC. Studies on the function of vitamin A in metabolism[J]. Biochem J, 1957, 225:995-1008. [11] Suano de Souza FI, Silverio Amancio OM, Saccardo Sarni RO, et al. Non-alcoholic fatty liver disease in overweight children and its relationship with retinol serum levels[J]. Int J Vitam Nutr Res, 2008, 78(1):27-32. [12] Musso G, Gambino R, De Michieli F, et al. Nitrosative stress predicts the presence and severity of nonalcoholic fatty liver at different stages of the development of insulin resistance and metabolic syndrome:possible role of vitamin A intake[J]. Am J Clin Nutr, 2007, 86(3):661-671. [13] Villa?a Chaves G, Pereira SE, Saboya CJ, et al. Non-alcoholic fatty liver disease and its relationship with the nutritional status of vitamin A in individuals with class III obesity[J]. Obes Surg, 2008, 18(4):378-385. [14] Kang HW, Bhimidi GR, Odom DP, et al. Altered lipid catabolism in the vitamin A deficient liver[J]. Molecular and Cellular Endoc-rinology, 2007, 271(1-2):18-27. [15]Oliveros LB, Domeniconi MA, Vega VA, et al. Vitamin A deficiency modifies lipid metabolism in rat liver[J]. Br J Nutr, 2007, 97(2):263-272. [16] Shiota G. Role of retinoic acid receptor in steatohepatitis related tumor formation[J]. J Gastroenterol Hepatol, 2007, 22(Suppl. 1):S101-107. [17] Amengual J, Ribot J, Bonet ML, et al. Retinoic acid treatment enhances lipid oxidation and inhibits lipid biosynthesis capacities in the liver of mice[J]. Cell Physiol Biochem, 2010, 25(6):657-666. [18] Liu AC, Lewis WG, Kay SA. Mammalian circadian signaling networks and therapeutic targets[J]. Nat Chem Biol, 2007, 3(10):630-639. [19] Navigatore-Fonzo LS, Golini RL, Ponce IT, et al. Retinoic acid receptors move in time with the clock in the hippocampus. Effect of a vitamin-A-deficient diet[J]. J Nutr Biochem, 2013, 24(5):859-867. [20] McNamara P, Seo SB, Rudic RD, et al. Regulation of CLOCK and MOP4 by nuclear hormone receptors in the vasculature:a humoral mechanism to reseta peripheral clock[J]. Cell, 2001, 105(7):877-889. [21] Golini RS, Delgado SM, Navigatore Fonzo LS, et al. Daily patterns of clock and cognition-related factors are modified in the hippocampus of vitamin A-deficient rats[J]. Hippocampus, 2012, 22(8):1720-1732. |